STEM Summer Research - RVC London Courses

You will earn 6 research credits over 6 weeks, conducting a faculty-supervised, hands-on, directed study research projects with results that will culminate in the preparation of a research paper. You will complete a minimum of 240 hours on research in and out of the laboratory.

Faculty mentors will work closely with you to direct your continued growth and knowledge development in the chosen research topic discipline.

  • Please review your project with your academic or study abroad advisor to ensure it will transfer back to your home school and that you are following your home school’s policies.

Choosing Your Research Project

  • Review Project titles and descriptions below.
  • List 3 (in order of preference) in your Academic Preferences Form, using LOND as the course code.
  • Program is highly individualized, with limited enrollment.
  • You will need to complete a brief Literature Review in consultation with your research supervisor prior to departure before the start of the program. More details here.
  • We encourage you to contact Arcadia Abroad Academic Advisors, Leah Cieniawa or Richard Evans II, to discuss your particular research interests further. 

Biomedical Sciences with the Royal Veterinary College, London

Course ID Title Credits Syllabus
LONS RSLW 392S International Independent Research in STEM Fields 6 PDF

Summer 2026 Research Projects

 

Regulation of lymphocyte gene expression by the transcription factor Ikaros

Supervisor: Dr Bradley Cobb

The Ikaros transcription factor is essential for lymphocyte development, yet its molecular function in regulating gene expression still remains largely unknown. This project will undertake a genetic approach by knocking out the Ikaros gene in a lymphocyte cell line using CRISPR technology then analysing the changes in expression of key lymphocyte genes.

Techniques: The student will learn many core techniques in molecular biology and protein biochemistry including PCR, recombinant DNA cloning, mammalian cell culture, and Western Blotting.

Two key literature references

  1. Heizmann, B., Kastner, P. & Chan, S. The Ikaros family in lymphocyte development. Current opinion in immunology 51, 14–23 (2017).
  2. Thompson, E. C. et al. Ikaros DNA-binding proteins as integral components of B cell developmental-stage-specific regulatory circuits. Immunity 26, 335–344 (2007).

Relevant Majors: Biological Sciences

 

Inflammation memory in tendon injury

Supervisor: Dr Debbie Guest

Tendon injuries occur commonly in horses. They undergo poor regeneration and have very high re-injury rates. Inflammation is believed to contribute to poor regeneration and this project will test the hypothesis that tendon cells can “remember” a previous injury-induced inflammatory event which predisposes them to re-injury.

Techniques: Cell culture, molecular biology (e.g. RNA extraction and quantitative PCR).

Two key literature references

  1. Beaumont RE, Smith EJ, Zhou L, Marr N, Thorpe CT, Guest DJ. Exogenous interleukin-1 beta stimulation regulates equine tenocyte function and gene expression in three-dimensional culture which can be rescued by pharmacological inhibition of interleukin 1 receptor, but not nuclear factor kappa B, signaling. Molecular and Cellular Biochemistry. 2023. doi: 10.1007/s11010-023-04779-z. 
  2. E. J. Smith, R. E. Beaumont, A. McClellan, C. Sze, E. Palomino Lago, L. Hazelgrove, J. Dudhia, R.K. Smith, D. J. Guest (2023) "Tumour necrosis factor alpha, Interleukin 1 beta and Interferon gamma have detrimental effects on equine tenocytes that cannot be rescued by IL-1RA or mesenchymal stromal cell-derived factors" Cell and Tissue Research, 391(3):523-544.

Relevant Majors: Biology, Molecular Biology 

 

The genetic contribution to fracture risk in Thoroughbred horses

Supervisor: Dr Debbie Guest

Fractures occur commonly in racing Thoroughbreds. The majority are caused by a failure of the bone to adapt to loading. Fracture is a complex condition with environmental and genetic risk factors. In this project we  will investigate how specific genes and DNA variants contribute to fracture risk.

Techniques: Cell culture, molecular biology (e.g. RNA extraction and quantitative PCR), bioinformatics.

Two key literature references: 1. Palomino Lago E, Ross AKC, McClellan A, Guest DJ. Identification of a global gene expression signature associated with the genetic risk of catastrophic fracture in iPSC-derived osteoblasts from Thoroughbred horses. Anim Genet. 2025 Feb;56(1):e13504. doi: 10.1111/age.13504. PMID: 39801206.

  1. E. Palomino Lago, A. Baird, S. C. Blott, R E McPhail, A. C. Ross, S.A. Durward-Akhurst, D. J Guest. A Functional Single-Nucleotide Polymorphism Upstream of the Collagen Type III Gene Is Associated with Catastrophic Fracture Risk in Thoroughbred Horses. Animals 2024, 14, 116. doi.org/10.3390/ani14010116.

Relevant Majors: Biology, Molecular Biology 

 

Identifying novel regulators of vascular calcification

Supervisor: Dr Isabel Orriss

Vascular calcification is a common consequence of aging and diseases such as type II diabetes and chronic kidney disease. Despite its prevalence there are currently no treatments to prevent or reverse vascular calcification. This project is part of our ongoing work to identify novel signalling systems that could be targeted therapeutically to treat this condition.

Techniques: Cell culture, biochemical assays, microscopy and imaging

Two key literature references:

  1. Bourne LE, Patel JJ, Davies BK, Neven E, Verhulst A, D’Haese PC, Wheeler-Jones CPD, Orriss IR. (2022). N-acetylcysteine (NAC) differentially affects arterial medial calcification and bone formation: the role of L-cysteine and hydrogen sulphide. J Cell Physiol 237:1070-1086.
  2. Bourne LE, Wheeler-Jones CPD, Orriss IR (2021). Regulation of mineralisation in bone and vascular tissue: a comparative review. J Endocrinol 248: R51-R56

Relevant Majors: Biology, Chemistry

 

Immune Gene Amplification in Wildlife Species: Understanding Genetic Diversity in Disease Resistance

Supervisor: Dr Eduard Roos

This project focuses on amplifying and analysing immune-related genes in various wildlife species to explore genetic diversity related to disease resistance. By studying these genes, we aim to gain insights into the evolutionary mechanisms of immune responses across species and their relevance to conservation biology and disease management.

Techniques:

  • PCR (Polymerase Chain Reaction)
  • Gel electrophoresis
  • DNA extraction and purification
  • Gene sequencing and bioinformatics analysis

Two key literature references:

  1. Acevedo-Whitehouse K., Gulland F.M., Greig D.J., Amos W. (2003) Disease susceptibility in California sea lions. Nature 422: 35-36.
  2. Sommer S. (2005) The importance of immune gene variability (MHC) in evolutionary ecology and conservation. Frontiers in Zoology 2: 16. 

Relevant Majors: Biology, Genetics, Veterinary Science, Conservation Biology

 

Understanding the role of Wnt signalling in cow embryo development and elongation

Supervisor: Dr Ali Fouladi

The Wnt/β-catenin signalling pathway is one of the typical signalling pathways that affects oocyte developmental competence and regulates embryo development in most species. One important feature of embryo development in some mammalian species including bovine is elongation of embryos to provide extended surface area for establishment of pregnancy and placentation. The actual mechanism of embryo elongation is not known. Interestingly, elongation of cow embryos does not happen during in vitro culture. Recent research has revealed the stimulatory effect of inhibiting Wnt signalling on proliferation of the trophectoderm (TE) cells of the blastocyst embryos. Using IVM/F/C techniques which are well-established in my lab and treatment of embryos with a negative regulator of Wnt signalling, this project aims to analyse the impact on TE cells growth and embryo elongation.

Techniques: Understanding reproductive cycle of cows, collecting oocytes from cow ovaries, assessment of oocyte quality, in vitro maturation and fertilisation and production of embryos (IVM/F/C), assessing embryo quality (morphological and after differential staining of ICM and TE cells in blastocysts). 

Two key literature references:

Zhang P, Zhang H, Li C, Yang B, Feng X, Cao J, Du W, Shahzad M, Khan A, Sun SC, Zhao X. Int J Mol Sci. 2024 Mar 31;25(7):3912. doi: 10.3390/ijms25073912

Amaral TF, Gonella-Diaza A, Heredia D, Melo GD, Estrada-Cortés E, Jensen LM, Pohler K, Hansen PJ. Biol Reprod. 2022 Oct 11;107(4):945-955. doi: 10.1093/biolre/ioac128.

Relevant Majors: Biology

 

Neuropilins as potential regulators of Epicardial to mesenchymal transition.

Supervisor: Dr Caroline Pellet-Many

The epicardium, the outermost layer of the heart, becomes activated following cardiac injury. During this response, epicardial cells proliferate and undergo an epithelial-to-mesenchymal transition (EpiMT). Using CRISPR-Cas9 gene editing, we generated epicardial cells deficient in neuropilins, receptors that are upregulated after zebrafish cardiac cryoinjury. Through qPCR and Western blot analyses, you will help us investigate the signaling pathways regulating EpiMT to determine whether neuropilins serve as key modulators of this process.

Techniques: Western blotting, qPCR, Fluorescent cell imaging

Two key literature references: 

  • Lowe V, Wisniewski L, Sayers J, Evans I, Frankel P, Mercader-Huber N, Zachary IC, Pellet-Many C. Neuropilin 1 mediates epicardial activation and revascularization in the regenerating zebrafish heart. Development. 2019 Jul 2;146(13):dev174482. doi: 10.1242/dev.174482. PMID: 31167777; PMCID: PMC6633600.
  • Wada AM, Smith TK, Osler ME, Reese DE, Bader DM. Epicardial/Mesothelial cell line retains vasculogenic potential of embryonic epicardium. Circ Res. 2003 Mar 21;92(5):525-31. doi: 10.1161/01.RES.0000060484.11032.0B. Epub 2003 Feb 6. PMID: 12600887.

Relevant Majors: Biological sciences

 

Investigating inflammatory mechanisms in kidney disease

Supervisor: Dr Elisavet Vasilopoulou

Kidney disease is often accompanied by an inflammatory response characterised by leukocyte infiltration and subsequent fibrosis, which results in kidney damage and correlates with declining renal function. This project will investigate inflammatory markers in healthy kidneys and kidneys affected by chronic kidney disease. 

Techniques: Immunohistochemistry, real-time PCR, imaging, data analysis.

Two key literature references: 

  • Vasilopoulou E, Riley PR, Long DA. (2018) Thymosin-b4: a key modifier of renal disease. Expert Opinion on Biological Therapy. 18(sup1):185-192 
  • Vasilopoulou E, Kolatsi-Joannou M, White KE, Robson MG, Sebire NJ, Riley PR, Winyard PJ, Long DA. (2016) Loss of endogenous thymosin-β4 accelerates glomerular disease. Kidney Int. 90(5): 1056-1070

Relevant Majors: Chemistry, Biology

 

Investigating age-related vascular degeneration in tendon

Supervisor: Dr Chavaunne Thorpe

Ageing-related tendon degeneration is common in horses and humans but is challenging to treat. Our research group aims to develop better treatments through understanding the contribution of microvascular cells to age-related tendon degeneration. To do this, we will investigate age-related changes in the vascular network within equine tendon tissues and employ immunofluorescence staining to visualise and characterise specific target proteins involved in vascular degeneration.

Techniques: Immunofluorescent staining, microscopy

Two key literature references: 

Relevant Majors: Biology, Biomedical Science

 

Investigating the mechanisms contributing to human vascular cell dysfunction in diabetes.

Supervisor: Dr Ashton Faulkner

The metabolic microenvironment associated with diabetes (hyperglycaemia/hyperlipidaemia) significantly impinges on vascular cell function in organs such as the heart and skeletal muscle, ultimately compromising their function. The molecular mechanisms underpinning this dysfunction are still not well established. This project will contribute to filling this knowledge gap through investigating mechanisms of vascular cell dysfunction driven by metabolic stress.  

Techniques: The student will have an opportunity to experience cell culture, molecular (qPCR), and biochemical (metabolic readouts) techniques, as well as the opportunity to develop skills in microscopy.

Two key literature references:

  • Faulkner, A. (2021). Trans-endothelial trafficking of metabolic substrates and its importance in cardio-metabolic disease. Biochemical Society Transactions, 49 (1): 507-517
  • Faulkner, A. et al. (2020). Dimethyl-2-oxoglutarate improves redox balance and mitochondrial function in muscle pericytes of individuals with diabetes mellitus. Diabetologia, 63 (10): 2205-2217

Relevant Majors: Biological Science, Biology, Chemistry


Grade Scale for the United Kingdom - AACRAO EDGE

The following information is vetted and provided by the American Association of Collegiate Registrars and Admissions Officers (AACRAO) on the Electronic Database for Global Education (EDGE).

Percentage Description U.S. Equivalent
70 – 100% First Class A
60 – 69% Second Class Upper B+
50 – 59% Second Class Lower B
40 – 49% Third Class/Pass C
0 – 39% Fail F
Intellectual property copyright AACRAO EDGE